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They included 29 case reports, and 23 clinical studies. Nonetheless, awareness of the adverse effect of certain pharmaceutical compounds on serum sodium concentrations facilitates a rational clinical management.Oxford University Press is a department of the University of Oxford.
Strategies to prevent this adverse drug effect involve careful consideration of risk factors and clinical and laboratory evaluation in the course of treatment. Exogenous vasopressin (ADH) is not effective due to the failure of the kidney to respond to ADH.
The most frequently reported causative agents were lithium (12 articles), followed by amphotericin (8 articles), vasopressin VHypernatraemia is a hyperosmolar state. Role of polydipsia and prostaglandinsSuccessful rescue of severe hypernatraemia (196 mmol/L) by treatment with hypotonic fluidLithium intoxication. Please check for further notifications by email. Hypernatremia in the neurologic intensive care unit: how high is too high?Hypernatremia in the intensive care unit: an indicator of quality of care?The changing pattern of hypernatremia in hospitalized childrenInterrelations between serum sodium concentration, serum osmolarity and total exchangeable sodium, total exchangeable potassium and total body waterHypernatremia in hospitalized patients: a sequel of inadvertent fluid administrationAppropriate treatment of hypernatraemia in diabetic hyperglycaemic hyperosmolar syndromeDisordered water channel expression and distribution in acquired nephrogenic diabetes insipidusEarly polyuria and urinary concentrating defect in potassium deprivationMolecular pathogenesis of nephrogenic diabetes insipidusMechanism of concentrating defect in hypercalcemia. independently searched for relevant articles in PubMed and EMBASE (up to February 2009) bibliographic databases. or by consensus.A total of 413 abstracts were screened, and 222 potentially relevant full-text articles were retrieved. Search for other works by this author on: Hyperosmolal states following cardiac arrestSevere hypernatremia after hypertonic saline irrigation of hydatid cystsFatal hypernatraemia after hypertonic saline irrigation of hepatic hydatid cystsHypocalcemic coma following two pediatric phosphate enemasSodium phosphate-induced hypernatremia in an elderly patient: a complex pathophysiologic stateFatal poisoning from sodium phosphate enema. Thank you for submitting a comment on this article. Renal causes of hypernatremia and volume depletion include therapy with diuretics. I don't think hypernatremia is a s/e of loop diuretics. George Liamis, Haralampos J. Milionis, Moses Elisaf, A review of drug-induced hypernatraemia, Drug-induced electrolyte abnormalities have been increasingly reported and may be associated with considerable morbidity and/or mortality. Moreover, ensuring adequate water ingestion is of paramount importance. In a series of 162 hypernatraemic patients, 13 developed hypernatraemia in the course of loop diuretic treatment [A recent 1:2 matched case–control study included 130 cases of hypernatraemia developed in the intensive care unit (ICU) compared to 260 controls.
It should be noticed that a combination of a very low sodium diet along with a thiazide diuretic (by diminishing the water delivery to the collecting tubules as a result of volume depletion-associated enhanced proximal NaCl and water reabsorption) as well as the usage of nonsteroidal anti-inflammatory drugs (possibly by normalizing the prostaglandin levels, which frequently are elevated in patients with NDI) may partially decrease urine volume [It is recommended that in patients with symptomatic hypernatraemia developed over a period of <48 h, a rapid correction of serum sodium concentration (1–2 mmol/L/h) should initially be performed, while a slower rate of serum sodium reduction (<0.5 mmol/L/h) should be attained in patients with hypernatraemia of longer or unknown duration [The amount of water necessary to correct hypernatraemia is estimated using the following equation: water requirement (L) = TBW × [(NaHypernatraemia may occasionally develop in the course of treatment with drugs used in everyday clinical practice (including lithium, lactulose, mannitol).
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