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Delayed sexual development: a study of 252 patients.
Philadelphia, Pa.: Lippincott Williams & Wilkins, 2005;401–64.Marshall WA, She received a medical degree from the University of Medicine and Dentistry of New Jersey-New Jersey Medical School in Newark. Dr. Master-Hunter completed a family practice residency at the University of Michigan Medical School.DIANA L. HEIMAN, M.D., C.A.Q., is assistant professor in the Department of Family Medicine at the University of Connecticut School of Medicine, Hartford. Müllerian agenesis: etiology, diagnosis, and management. Secondary amenorrhea is more common than primary amenorrhea.A female patient with primary amenorrhea and sexual development, including pubic hair, should be evaluated for the presence of a uterus and vagina.Women with secondary amenorrhea should receive pregnancy tests.Women with polycystic ovary syndrome should be tested for glucose intolerance.A female patient with primary amenorrhea and sexual development, including pubic hair, should be evaluated for the presence of a uterus and vagina.Women with secondary amenorrhea should receive pregnancy tests.Women with polycystic ovary syndrome should be tested for glucose intolerance.Pubertal changes typically occur over a three-year period and can be measured using Tanner staging.Physicians should conduct a comprehensive patient history and a thorough physical examination of patients with amenorrhea Exercise, weight loss, current or previous chronic illness, illicit drug usePrevious central nervous system chemotherapy or radiationPsychosocial stressors; nutritional and exercise historyMenarche and menstrual history (mother and sisters)Dysmorphic features (e.g., webbed neck, short stature, widely spaced nipples)Striae, buffalo hump, significant central obesity, easy bruising, hypertension, or proximal muscle weaknessUndescended testes; external genital appearance; pubic hairSigns and symptoms of hypothyroidism or hyperthyroidismExercise, weight loss, current or previous chronic illness, illicit drug usePrevious central nervous system chemotherapy or radiationPsychosocial stressors; nutritional and exercise historyMenarche and menstrual history (mother and sisters)Dysmorphic features (e.g., webbed neck, short stature, widely spaced nipples)Striae, buffalo hump, significant central obesity, easy bruising, hypertension, or proximal muscle weaknessUndescended testes; external genital appearance; pubic hairSigns and symptoms of hypothyroidism or hyperthyroidismAlgorithm for the evaluation of primary amenorrhea.
Snow-Harter C,
Hergenroeder AC, Menstrual dysfunction in anorexia nervosa. Schwenk TL. 6th ed. (FSH = follicle-stimulating hormone; LH = luteinizing hormone. Hier sollte eine Beschreibung angezeigt werden, diese Seite lässt dies jedoch nicht zu. Variations in patterns of pubertal changes in girls. Secondary amenorrhea is the absence of menses for three months in women with previously normal menstruation and for nine months in women with previous oligomenorrhea. Drinkwater BL, Bromocriptine (Parlodel) is effective, but cabergoline (Dostinex) has been shown to be superior in effectiveness and tolerability.Two common causes of normogonadotropic amenorrhea are outflow tract obstruction and hyperandrogenic chronic anovulation. (TSH = thyroid-stimulating hormone; MRI = magnetic resonance imaging; FSH = follicle-stimulating hormone; LH = luteinizing hormone. Microadenomas are slow growing and rarely malignant. Nelson LM. Miller KK, Bone mineral density after resumption of menses in amenorrheic athletes.
Constitutional delay of growth and puberty commonly causes primary amenorrhea in patients with no sexual development.
Diagnosis and management of pituitary tumors: recent advances. (TSH = thyroid-stimulating hormone; MRI = magnetic resonance imaging; FSH = follicle-stimulating hormone; LH = luteinizing hormone. Constitutional delay of growth and puberty commonly causes primary amenorrhea in patients with no sexual development. Dr. Master-Hunter completed a family practice residency at the University of Michigan Medical School.DIANA L. HEIMAN, M.D., C.A.Q., is assistant professor in the Department of Family Medicine at the University of Connecticut School of Medicine, Hartford. The most common cause of outflow obstruction in secondary amenorrhea is Asherman’s syndrome (intrauterine synechiae and scarring, usually from curettage or infection).Polycystic ovary syndrome (PCOS) is the most common cause of hyperandrogenic chronic anovulation. Modest weight loss can lower androgen levels, improve hirsutism, normalize menses, and decrease insulin resistance.
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